Pathophysiology of venous stasis ulcers and Deep vein thrombosis.

To complete:
Write a 2- to 3-page paper that addresses the following:
�Compare the pathophysiology of chronic venous insufficiency and deep venous thrombosis. Describe
how venous thrombosis is different from arterial thrombosis.
�Explain how the patient factor you selected might impact the pathophysiology of CVI and DVT.
Describe how you would diagnose and prescribe treatment of these disorders for a patient based on the
factor you selected.
�Construct two mind maps-one for chronic venous insufficiency and one for deep venous thrombosis.
Include the epidemiology, pathophysiology, and clinical presentation, as well as the diagnosis and
treatment you explained in your paper.

Pathophysiology of venous stasis ulcers and Deep vein thrombosis
Chronic Venous insufficiency (CVI) vs. Deep Vein thrombosis (DVT)

Running Head: Pathophysiology of venous stasis ulcers and Deep vein thrombosis
Chronic Venous insufficiency occurs when the valves in the venous system malfunction.
This is facilitated by various incidences such as age, smoking, and increased physical
inactiveness. When the valves are damaged, they cause the blood to flow back. This forms blood
clot in the vein found along the hind limb, causing a disease condition known as Deep Vein
Thrombosis (DVT). The clinical manifestation of the disease includes swelling of the ankle if the
patient stands of sits for a long period. The swelling is sometimes painful and causes flaking of
the skin and itchiness (Davies, Lumsden, & Vykoukal, 2011). Deep Vein Thrombosis (DVT) is a
cardiovascular complication that results when the venous valves located at the hind limb
becomes damaged. The venous system transports deoxygenated blood from the rest of the body
to the heart for oxygenation. During this cycle, blood flows upwards from legs to the heart. For
this to occur, the muscles of the feet and the calf contract, squeezing the veins which then
propel blood upward towards the heart. The vein has valves are aligned in one direction and
facilitate blood transportation. Consequently, blood circulation is disturbed, making it difficult
for blood to circulate effectively and blood pools in these veins (Kibbe, Pearce, & Yao, 2010).
CVI and DVT compare in that the diagnostic process is similar and includes use of
duplex ultrasound technology to check the blood circulation process. The technique applied is as
follows, the transducer part of the machine is normally placed on the site that s directly above
the vein in question, the waves on how the blood is flowing together with a 3D formulation of
the vein anatomical structure is displayed on the monitor. This diagnosis is important because it
is able to diagnose early enough, and the disease gets treated using the appropriate medication.
The pharmacological medication is often coupled with on pharmacological medication, such as
weight loss, exercises, and the reduction of environmental stressors such as standing for a long

Running Head: Pathophysiology of venous stasis ulcers and Deep vein thrombosis
period. Other measures include compression stockings and taking antibiotic (Huether, &
McCance, 2012). 
Evidence based research indicates that the CVI and DVT prevalence illustrate healthcare
disparity. This is mainly due to the interactions of healthcare determinants such as genetic,
environmental factors, ethnic background, and socioeconomic status. The prevalence rate is high
among the African American, Hispanics, and Caucasians as compared to White. Literature
associates these findings with environmental exposures and socio economic background. For
instance, the white population has high rates of educated people and low rates of low household
income (Kibbe, Pearce, & Yao, 2010). This implies that they are able to access the hospital
routinely and are treated early before the disease clinical manifestations become worse. On the
other hand, these other communities have the highest number of dropouts. The children are more
susceptible to drugs and poor living condition at a tender age. Additionally, the high hospital cost
act as an obstacle to accessing quality care. Therefore, these people will really on alternative
therapies and over counter drugs for a long period, and will only access care during the last stage
of the disease. These communities have the highest rates of obesity, which is a main risk factor
to DVT and CVI (Murphy & Lloyd, 2013).

Running Head: Pathophysiology of venous stasis ulcers and Deep vein thrombosis
Davies, M., Lumsden, A., & Vykoukal, D. (2011). Chronic venous insufficiency. Minneapolis:
Cardiotext Pub.
Huether, S. E., & McCance, K. L. (2012). Understanding pathophysiology (Laureate custom
ed.). St. Louis
Kibbe, M., Pearce, W., & Yao, J. (2010). Venous disorders. Shelton, Conn.: People’s Medical
Pub. House—USA.
Murphy, J., & Lloyd, M. (2013). Mayo Clinic cardiology. Oxford: Mayo Clinic Scientific
Press/Oxford University Press.

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