Pneumothorax Case Study

Explain to the
pathophysiology of a pneumothorax and the UWSD and how it affects the cardiac &
respiratory system using the correct medical and nursing terminology. the Assessment criteria
below must be met in regards to this.
Assessment Criteria.

  1. We are to give a detailed explanation of the pathophysiology of the illness/disease of the
    case patient, MRS Richards.
  2. Explanation of 2 signs/symptoms the case patient presented with and why they manifested
    as part of the illness/disease. The two sign and symptom Mrs Richards had presented were
    chest tightness and shortness of breath.
  3. Critical thinking and rationale – The patient, Mrs. Richards, is on some Antibiotics
    (Metronidazol
    e and Cephalothin). Link these medication, A&P, pathophysiology, context and
    theory together and give detail description as to why the medication is being given to Mrs
    Richards, in regards to her pneumothorax, chest drain (UWSD).
    Lastly linking pharmacology to multiple parts of practice such as; infection control, safety,
    Nursing con

Pneumothorax Case Study

Introduction
Pneumothorax is a medical condition where the pleural cavity gets filled of air, which
impairs ventilation and oxygenation. This health complication occurs when air leaks in the
pleural cavity (space between lungs and chest walls) which creates an external force outside
(destroys negative pressure) the lungs making them to collapse. It can be caused by chest
injury or medical health disorders. This medical complication may occur in various clinical
settings and in any person of any age (Tamburrini, Sellitri, Tacconi, Brancati & Mineo,
2015). This paper explores the disease pathophysiology and ways it with the disease clinical

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manifestation. The paper also critically analyses the medication to give detailed information
on the rationale behind the administration of specific medication. The concepts of
pharmacology will be linked to nursing practice such as patient safety and infection control.
Causes and risk factors
Pneumothorax is caused by many factors. To start with, the patient may experience
chest injuries (blunt or penetrating) that occur during physical accidents or physical assaults.
Others causes include pleural lacerations caused during the medical procedures such as
inserting aspiration needles in the patient chest. Damaged lung tissues are likely to cause air
leakages into the pleural cavity. Lung disorders can be caused by many underlying diseases
such as pneumonia, cyctic fibrosis or chronic pulmonary diseases. In some cases, small air
blisters (blebs) can develop in the lungs, which could burst, causing air to leak into the spaces
surrounding the lungs. There incidences when pneumothorax occur in patients with
breathing difficulty and who require mechanical assistance. The mechanical ventilation used
can cause imbalance of air pressure and could cause the lungs to collapse (Tamburrini,
Sellitri, Tacconi, Brancati & Mineo, 2015).
The main risk factors associated with pneumothorax includes gender, where men are
more likely to suffer from pneumothorax as compared to women. The risk of pneumothorax
increases with poor lifestyles such as smoking. Pneumothorax caused by ruptured air blisters
is common in people between the ages of 20 years and 40 years, and common in tall and
underweight people. Genetics also plays a huge role as it mainly appears in families with a
history of pneumothorax. Other risk factor includes lung disorders and mechanical
ventilation (Ling, Wu, Ming, Cai & Chen, 2015).
Pathophysiology and clinical manifestation
The pathophysiology of pneumothorax is described according to the causes.
Traumatic pneumothorax occurs due to penetrative injuries such as impaled object, gunshot

Pneumothorax Case study 3
wound, and stab wound which are associated with traumatic open pneumothorax, closed
pneumothorax or heamo-thorax of the pneumothorax. Traumatic open pneumothorax occurs
when the pleura gets lacerated and allows accumulation of atmospheric air in the pleural
cavity. It mainly cause by a penetrating chest trauma. Air in the pleural cavity interrupts
normal negative pressure which makes the lungs not remain inflated. The traumatic closed
pneumothorax occurs when laceration of the pleura is caused by a blunt trauma, causing air to
enter the pleural cavity (Tamburrini, Sellitri, Tacconi, Brancati & Mineo, 2015).
Latrogenic traumatic pneumothorax occurs when the pleura laceration is caused by
therapeutic and diagnostic procedures such as needle biopsy, mechanical ventilation, central
line insertion, and aspirations. Air trapped in pleural cavity interrupts normal negative
pressure which makes the lungs not remain inflated. Spontaneous pneumothorax is an
example of closed type. This is common in geriatric patients diagnosed with chronic
pulmonary diseases but can also occur in adults. The main cause of pleura rapture is mainly
underlying respiratory disorders such as asthma, pneumonia, chronic air flow limitation
(CAL), tuberculosis, disorders of the connective tissues, Cystic fibrosis and Marfan’s
syndrome.
Tension pneumothorax is caused by either traumatic of spontaneous pneumothorax.
The pleura get raptured allowing air into the pleura cavity but do not allow it to escape. This
creates tension and pressure in the intrapleural space, which makes the affected lung to
collapse, squeezing the mediastinal contents to the unaffected lung. Air in the pleural cavity
interrupts normal negative pressure which makes the lungs not remain inflated (Corsini,
Pratesi & Dani, 2013).
The main clinical signs of pneumothorax are the sudden sharp pain around the chest,
which is normally exacerbated by chest movements such as coughing and breathing. The
asymmetrical chest movement causes the lungs to collapse. These results to cyanosis and

Pneumothorax Case study 4
shortness of breath associated with hypoxia. Other clinical manifestations include decreased
vocal fremitus and respiratory distress, which are associated with collapsing of the lungs.
When auscultation is done, the breath sounds on the side affected lung are absent as the lung
has collapsed (Chiu, Chen, Wang, Tsai & Wong, 2014).
Chest rigidity on the affected side may occur due to tachycardia and hypoxia.
Palpation of the affected side normally reveals crackling sound just beneath the skin, which is
normally due to leaking of air into the tissues. Research indicates that Tension Pneumothorax
has the most severe clinical manifestation which includes hypotension, reduced cardiac
output, tachypnea and compensatory tachypnea. The disease diagnosis is normally done
using X-rays, which is normally reveals air in the pleural space. Arterial blood analysis
normally can be used to reveal hypoxemia (Lee et al., 2014).
Medical management and patient education
The main medical management is done by cutting through the chest (thoracotomy) is
required. This is done by inserting of a chest tube in the 5 th and 6 th intercostal space located
under the patient’s arm. The tube is then attached to a water-seal drainage system, which
drains all the air in the pleural cavity. The chest tube is normally a long hollow tube which is
inserted as described above. This is normally done under local anaesthesia (Gale et al., 2015).
The pleural space refers to the space between the visceral and parietal layers that line has
inside the lungs and chest cavity. Normally, the two layers are held together by negative
pressure or vacuum, which ensures that the lungs remain inflated. The pleural space is
normally empty except the small amount of fluid that helps reduce friction between the
cavities. In special cases, air or fluid may accumulate causing pneumonia, chest trauma or
other intraabdominal conditions. Pneumothorax could also occur. This results to chest
tightness and breathing difficulty (Baird & Gandhi, 2014).

Pneumothorax Case study 5
The chest drains are done so long as air remains in the pleural cavity. This must be
done because without UWSD, pneumothorax can result into tension pneumothorax. This is
because the air trapped in the pleural cavity prohibits air outflow. Consequently, the volume
of the air in the pleural cavity increases with every inspiration causing the pressure in the
pleural cavity to increase causing lungs to collapse. Further increase of pressure in the pleural
cavity makes the mediastinum shift towards the contralateral side of the lungs, which
obstructs the venous return to the heart. This causes circulatory instability and could result
into a traumatic arrest. This is indicated by cardiovascular diseases, reduced oxygen and low
blood pressure. The patient needs to have regular chest X-rays and to monitor the disease
progress (Curfman, Robitsek, Sammett & Schubl, 2015).
This intensive care is associated with potential risks and complications even when
performed by experienced staff. The main common health complications are pain and
infection. For example, drains inserted into the body increases the risks of infection.
Therefore, the patients and patient relatives must not touch the drain, tubes and the dressings
that cover the site of insertion. Good hygiene practices such as washing hands before and
after contacting patient also helps minimize patient’s risks of infection. Some degree of pain,
infection control and patient safety must be discussed by the healthcare providers (AL Saif,
Hammodi, Al-Azem & Al-Hubail, 2015).
The treatment of pneumothorax mainly depends on the size of the pneumothorax.
Small pneumothorax normally resolves by its self. The aim of the treatment is to get rid of the
accumulated air in the pleural space. Once the lungs are filled with air, the patient is able to
breath with ease. The patient is given Metronidazole and Cephalothin antibiotics in order to
treat and prevent bacterial infection during UWSD procedures. The patient should also be
given pain medication such as Tylenol. The pain prescription is done to decrease patient’s
pain. The pain medication should be done as directed. Sedative medication are given to make

Pneumothorax Case study 6
the patient remain calm and relaxed when performing needle aspirations and chest tubes
(Dixit, Meena & Patil, 2015).

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References

AL Saif, N., Hammodi, A., Al-Azem, M., & Al-Hubail, R. (2015). Tension Pneumothorax
and Subcutaneous Emphysema Complicating Insertion of Nasogastric Tube. Case
Reports in Critical Care, 2015, 1-4.
Baird, A., & Gandhi, M. (2014). Pneumopericardium and pneumothorax due to right atrial
permanent pacemaker lead perforation. Journal of Medical Imaging and Radiation
Oncology, 59(1), 74-76.
Chiu, C., Chen, T., Wang, C., Tsai, M., & Wong, K. (2014). Factors associated with
proceeding to surgical intervention and recurrence of primary spontaneous
pneumothorax in adolescent patients. European Journal of Pediatrics, 173(11), 1483-
Corsini, I., Pratesi, S., & Dani, C. (2013). Pulmonary interstitial emphysema after resolution
of relapsing pneumothorax and discontinuation of mechanical ventilation. An atypical
case in a preterm infant. The Journal of Maternal-Fetal & Neonatal Medicine, 27(15),
1610-1612.
Curfman, K., Robitsek, R., Sammett, D., & Schubl, S. (2015). Blunt trauma resulting in
pneumothorax with progression to pneumoperitoneum: a unique diagnosis with
predicament in management. J. Surg. Case Rep., 2015(12), rjv147.

Dixit, R., Meena, M., & Patil, C. (2015). Pneumomediastinum, bilateral pneumothorax and
subcutaneous emphysema complicating acute silicosis. Int J Occup Med Environ
Health, 2015(2).
Gale, M., Loarte, P., Mirrer, B., Mallet, T., Salciccioli, L., Petrie, A., & Cohen, R. (2015).

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Takotsubo Cardiomyopathy in the Setting of Tension Pneumothorax. Case Reports in
Critical Care, 2015, 1-5.
Lee, K., Kim, K., Kim, E., Lee, J., Kim, Y., & Hyun, S. et al. (2014). Detection of blebs and
bullae in patients with primary spontaneous pneumothorax by multi-detector CT
reconstruction using different slice thicknesses. Journal of Medical Imaging and
Radiation Oncology, 58(6), 663-667.
Ling, Z., Wu, Y., Ming, M., Cai, S., & Chen, Y. (2015). The Effect of Pleural Abrasion on
the Treatment of Primary Spontaneous Pneumothorax: A Systematic Review of
Randomized Controlled Trials. PLOS ONE, 10(6), e0127857.
Tamburrini, A., Sellitri, F., Tacconi, F., Brancati, F., & Mineo, T. (2015). Simultaneous
Bilateral Spontaneous Pneumothorax Revealed Birt-Hogg-Dubè Syndrome.

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